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But when the scientists started playing around in the lab, they realized it didn't just stop fungal cells from growing it also stopped human cells.
The way that rapamycin works is it targets a fundamental central component of cellular metabolism.
It starts this process called autophagy, which is Greek for self-eating, "auto-phagy." And that means it consumes old, damaged proteins and then recycles them into fresh, new ones.
Listen to native speakers pronounce “rapamycin” in real conversational contexts with synchronized timestamps and subtitles.